ittle or no
pharmacological effect, may become dangerous in the presence of
an MAO inhibitor and cause headache, stiff neck, cardiovascular
difficulties, and even death. MAO inhibitors may intensify and
prolong the effects of other drugs (CNS depressants, narcotic
analgesics, anticholinergics, dibenzazepine antidepressants,
etc.
) by interfering with their metabolism. In the presence of an
MAO inhibitor many substances which are ordinarily non-active
because of their swift metabolism may become potent psychoactive
drugs. The phenomenon may create a new series of mind alterants.
file:///C|/Documents%20and%20Settings/All%20Users/...0Culture/Ayahuasca%20and%20DMT/DMT%20FAQ%202.0.txt (4 of 13)4/14/2004 9:11:34 PM
file:///C|/Documents%20and%20Settings/All%20Users/Documents/Bo.
.
.
%20Counter%20Culture/Ayahuasca%20and%20DMT/DMT%20FAQ%202.
0.
txt
However, because of the complex and precarious variables
involved, it is risky and foolish for anyone to experiment with
these possibilities on the non-professional level.
The most commonly used MAO inhibitors include hydrazines
such as iproniazid, Marsilid, Marplan, Niamid, Nardil, Catron;
also non-hydrazines such as propargylamines, cyclopropylamines,
aminopyrazine derivatives, indolealkylamines, and
carbolines.
MAO-inhibiting materials discussed in this book
include yohimbine, various tryptamines, especially 5-MeO-DMT and
the methyltryptamines, and the various harmala alkaloids. The
latter are especially potent inhibitors but, like yohimbine and
the trytamines, are short)lasting in action (30 minutes to
several hours). Some of the commercial MAO inhibitors listed
above are effective for several days to several weeks.
Among the materials which may be dangerous in
combination with MAO inhibitors are sedatives, tranquilizers,
antihistamines, narcotics and alcohol ) any of which can cause
hypotensive crisis (severe blood pressure drop); and amphetamines
(even diet pills), mescaline, asarone, nutmeg (active doses),
macromerine, ephedrine, oils of dill, parsley or wild fennel,
beer, wine, cocoa, aged cheese and other tyrosine)containing
foods (tyrosine is converted into tyramine by bacteria in the
bowel) ) any of which can cause hypertensive crises (severe blood
pressure rise).
SYRIAN RUE Peganum harmala. Family Zygophyllaceae (Caltrop
family.)
Material: Seeds of woody perennial native to Middle East.
(Roots also active but seldom used.
)
Usage 1 oz. seeds are thoroughly chewed and swallowed. Most
effective when combined with other psychotropic materials,
especially those containing tropanes.
Active Constituents: Harmine, harmaline and harmalol.
Effects and Contraindications: Hallucinogen; see harmine.
HARMINE 7-methoxy-1-methyl-9H-pyrido (3,4-b) indole.
Material: Indole-based alkaloid found in several plants
including Banisteriopsis caapi (from which the South American
hallucinogenic brew yage is prepared), Peganum harmala (Syrian
file:///C|/Documents%20and%20Settings/Allittle or no
pharmacological effect, may become dangerous in the presence of
an MAO inhibitor and cause headache, stiff neck, cardiovascular
difficulties, and even death. MAO inhibitors may intensify and
prolong the effects of
Extasyephedra other drugs (CNS depressants, narcotic
analgesics, anticholinergics, dibenzazepine antidepressants,
etc.) by interfering with their metabolism. In the presence of an
MAO inhibitor many substances which are ordinarily non-active
because of their swift metabolism may become potent psychoactive
drugs. The phenomenon may create a new series of mind alterants.
file:///C/Documents%20and%20Settings/All%20Users/...0Culture/Ayahuasca%20and%20DMT/DMT%20FAQ%202.0.txt (4 of 13)4/14/2004 9:11:34 PM
file:///C/Documents%20and%20Settings/All%20Users/Documents/Bo...%20Counter%20Culture/Ayahuasca%20and%20DMT/DMT%20FAQ%202.0.txt
However, because of the complex and precarious variables
involved, it is risky and
Zone Diet foolish for anyone to experiment with
these possibilities on the non-professional level.
The most commonly used MAO inhibitors include hydrazines
such as iproniazid, Marsilid, Marplan, Niamid, Nardil, Catron;
also non-hydrazines such as propargylamines, cyclopropylamines,
aminopyrazine derivatives, indolealkylamines, and
carbolines. MAO-inhibiting materials discussed in this book
include yohimbine, various tryptamines, especially 5-MeO-DMT and
the methyltryptamines, and the various harmala alkaloids. The
latter are especially potent inhibitors but, like yohimbine and
the trytamines, are short)lasting in action (30 minutes to
several hours). Some of the commercial MAO inhibitors listed
above are effective for several days to several weeks.
Among the materials which may be dangerous in
combination with MAO inhibitors are sedatives, tranquilizers,
antihistamines, narcotics and alcohol ) any of which can cause
hypotensive crisis (severe blood pressure drop); and amphetamines
(even diet pills), mescaline, asarone, nutmeg (active doses),
macromerine, ephedrine, oils of dill, parsley or wild fennel,
beer, wine, cocoa, aged cheese and other tyrosine)containing
foods (tyrosine is converted into tyramine by bacteria in the
bowel) ) any of which can cause hypertensive crises (severe blood
pressure rise).
SYRIAN RUE Peganum harmala. Family Zygophyllaceae (Caltrop
family.)
Material: Seeds of woody perennial native to Middle East.
(Roots also active but seldom used.)
Usage 1 oz. seeds are thoroughly chewed and swallowed. Most
effective when combined with other psychotropic materials,
especially those containing tropanes.
Active Constituents: Harmine, harmaline and harmalol.
Effects and Contraindications: Hallucinogen; see harmine.
HARMINE 7-methoxy-1-methyl-9H-pyrido (3,4-b) indole.
Material: Indole-based alkaloid found in several plants
including Banisteriopsis caapi (from which the South American
hallucinogenic brew yage is prepared), Peganum harmala (Syrian
file:///C/Documents%20and%20Settings/All
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